Hey Seun. Not sure if a deacon break is needed. I’m thinking what is needed for further progress is for me to find the right dosage to stay above a 75% erection level for 2 to 4 hours. What’s happening right now is pge1 by itself is not keeping me above 75% hardness for over 2 to 4 hours so biomechanical creep can take place as tunica delinking is going on. As soon as I get my other vasodilators to help me get my erection time in, I’m confident that things will improve.
Hey bigdex28. Good find on the info bro.
Hey Syd Kitten. Yeah I know. Example 1 made the biggest gains out of all 10 test subjects. Going from 5 inches to over 9 inches is a huge gain in 18 months time.
Hey dickerschwanz. The thing is I use to pump right after my pge1 erections would subside which is extremely painful do to post pge1 soreness. Now that I’m injecting almost 100 mcgs of pge1 the pain is way too much for me to pump during or right after. Not a chance in hell unless I’m drugged up on strong pain blockers or something. LOL.
I’m am starting to agree with you about chemical Penis Enlargement being the erection time being more important then delinking properties of pge1. If it turns out that largest doses of pge1 isn’t needed and it is indeed the erection time that really matters then other vasodilators can work to achieve a 2 to 5 hour erection without pain.
I could just use 50 mcgs of pge1 which will bring manageable pain then find the right dosage of phentolamine and forsklin to stack with it to bring me up to the 3plus hours of erection at above 75% hardness time mark for better progress.
Hey agent7x6. Yeah examples 5 and 6 did use dht and example 10 was actually getting dht injected into his penis with the vasodilators.
Hey MikeShlort. Here’s a link showing the benefits of pge1 aka prostaglandin.
http://www.ncbi.nlm.nih.gov/puBathmateed/7861547
When I say delinking I mean literally that pge1 unwoven and unravel the cross sectional collagen bonds of the tunica at microscopic levels. This is a process that takes months. As more collagen is unwoven under internal stress which would be the stress of the erection itself and other forms of Penis Enlargement, the tunica will start to give into the stress and enlarge through deformation and mechanical creep. As deformation takes place new cells are generated to fill in the gaps making the tunica sheath that covers the CC and the CS longer which means more blood can engorge the CC and CS before the tunica pressurizes and becomes 100% rigid meaning full erection.
When this happens you have enlargement. Keep repeating this over months to years and the tunica will continue to unweave and remodel giving more and more slack so more blood can engorge the penis before the tunica pressurizes into a full rigid erection. With pge1’s unraveling effects this causes the growth of the tunica through stretch force by expanding the sheath and not the thickness which is what we want because if the thickness of the tunica through cross sectional collagen synthesis builds up then it will make it harder to make gains. AKA TOUGH TUNICA SYNDROME. That’s why the pge1 vasodilator, tb-500 and the DMSO/PABA are used to prevent this.
Here are some quotes in the detailed description of invention in Dr Adam’s patent showing pge1’s aka prostaglandin’s delinking effects. He also talks about the use of relaxin as a potentiator as well.
“0055] Relaxin directly and indirectly triggers a cascade of complex biochemical and cellular effects that can cause general morphological changes to genitalia. Prostaglandins such as prostaglandin F2 alpha and prostaglandin E2 have similar effects. This invention includes the mediators of these cascades as potentiators”.
[0043]A prostaglandin potentiator can be prostaglandin F2 alpha or prostaglandin E2. The potentiator might be relaxin, prostaglandin F2 alpha, or prostaglandin E2, or the biochemical mediators that result in the desired changes in collagen or the connective tissue that produces and remodels collagen and express the effects of relaxin, prostaglandin F2 alpha, or prostaglandin E2.
[0054]The potentiator may be a pharmacological agent or combination of agents that promote cellular processes that result in biological and/or mechanical creep and ultimately induce remodeling of the connective tissues that help define the size and shape of the penis. In addition, an agent which increases solubility of collagen may be used as a potentiator. Agents with very specific mechanisms of action may be used, or other agents with pleomorphic mechanisms of action, such as relaxin or growth hormone which trigger diverse mechanisms to induce growth in the penis may be used. For example, agents may be administered that facilitate the elongation of collagen fibers and accelerate the turnover remodeling rates of collagen through numerous mechanisms. For example, D-penicillamine and dimethyl sulfoxide (DMSO), which promote the elongation of collagen by inhibiting or interfering with inter- and intramolecular collagen cross-linkage may be used. Other agents include, but are not limited to, relaxin, insulin like growth factors, growth hormone, metalloproteinases or metalloproteinases agonists or promoters of collagenase activity, tissue inhibitors of matrix metalloprotenases (TIMPs) other agents that increase collagen solubility, prostaglandins, corticosteroids, or aminobenzoate potassium, a commercial brand being known as Potaba®. Preferred prostaglandins are prostaglandin F2 alpha and prostaglandin E2. Also included are pharmaceutically active sequences, peptidomimetics, or mimetics above the above-listed molecules.
Hey Mikeshlorts. With you saying to just use pge1 for 18 months keep in mind that I’ve been using pge1 now for over 5 months and have reached a point to here I need high dosages to have a good 2 to 4 hour erection if im not injecting close to bedtime. Right now at 90 mcgs it’s not getting me there and the pain is crazy with that much pge1. Not to mention at this point I can’t fit any more pge1 in a 1 CC syringe. I either need to find a source for a higher pge1 concentration or just use other stronger vasodilators to stack with a smaller amount of pge1 to reach 2 to 4 hour erections again like what was done in the patent and what Ronielle did after he reached 500 mcgs of pge1.
Keep in mind that yes there are a lot of theories going around but that’s the point of this thread. What Im doing is experimenting on myself to see what’s the optimal way of aheaving enlargement. So far I’ve gained a quarter inch in midgirth and base girth so this does work even for a vet like me. Right now I’m leaning towards the using what ever safe vasodilator necessary to achieve a nice tunica stretching 2 to 5 hour erection and starting to lean away from the goal of reaching 500 mcgs of PAINFUL pge1 like some of the test subjects did and Ronielle did.